Updates for November 2011

Updates for November 2011

Key
B=Bacteria D=Disease F=Fungus P=Parasite V=Virus	C=Chemical E=Emotional M=Miasm* T=Toxins
*Multiple letters indicate a combination of issues

Name	Symptom Counter Vibration	Bottles	Contents	Description
ARG LYS	ARGININOSUCCINIC ACID LYSASE			Developed to help the reversal of Urea Cycle Disorders of Arginosuccinase Acid Lyase.
ARGNIN	ARGININE			Developed to help the reversal of Urea Cycle Disorders of Argininosuccinic Acid Synthetase, Ornathine Transcarbamylase Deficiency,
ARGNSE	ARGINASE			Developed to help the reversal of Urea Cycle Disorders of Arginase
BIT SFR	BITE STING FRESH		T	After taking Bite Sting complex and clearing the miasms of poison, it is better to use this full-strength antidote for new, fresh bites or stings.
CITRLN	CITRULLINE			Developed to help the reversal of Urea Cycle Disorders of Argininosuccinic Acid Synthetase, Ornathine Transcarbamylase Deficiency,
EMB C CX	EMBRYO CELL COMPLEX		MVB	Embryo Cell Cancer is the most common form of prostate cancer recognized. The formula contains the disease, lump, carcinoma and retrovirus.
GBL C CX	GOBLET CELL COMPLEX		MVB	Goblet cells are glandular simple columnar epithelial cells whose sole function is to secrete mucin, which dissolves in water to form mucus. They are found scattered among the epithelial lining of organs, such as the intestinal and respiratory tracts. They are found inside the trachea, bronchus, and larger bronchioles in respiratory tract, small intestines, the colon, peritoneum and conjunctiva in the upper eyelid. In this disease they form too much mucous and choke breathing as well as digestion. There is some evidence they play a role in Pseudomyxoma Peritonei.
HYDR GP	HYDROCELE GROUP		MVB	Hydroceles are caused by many histamines flooding an infected cell and filling it so much that the stretched cell walls close the exits and the cell remains stuffed with dying histamines. The cell is so crowded that it cannot function and creates stagnation of cell activity. The first cell so identified was the intestinal pericytes (not to be confused with paristies). When there is intestinal infection, the pericytes are infected, fill with histamines, create a hydrocele and prevent the production of Chloride Adenosine Triphosphate Glutamate, the nerve electrolytes. The nerves never receive the needed electrolytes and cannot regain function even after the original cause of nerve dysfunction is removed.
NCRB GL	N- CARBAMYL-GLUTAMATE			Developed to help the reversal of Urea Cycle Disorders of Carbamoyl Phosphate Synthetase and N- Acetylglutamate Synthetase.
PYROL	PYROLURIA		MVF	This disease is associated with high levels of Hydroxyhemppyrolin-2-one in the urine. (That name is enough to dissuade many from even looking at the disease). It precipitates a severe deficiency of zinc and vitamin B6 (and often Vitamin B3) which are lost in urine. The list of symptoms include a number of blocks to full potential in thinking, personality, digestion, pessimism, withdrawal and anger (to name a few). The first discoveries were made in the 1950s. There have been changes in names, confusion, and lousy tests for confirmation. Medicine rejected it as a disease while the vitamin/mineral folks have made substantial improvements in life quality with large doses of the lost vitamins and zinc. There remains controversy whether this is caused by infection or genetics. We find it is genetic and present this remedy as an opportunity to be rid of the disease instead of constantly taking supplements.
TRD LK1	THYROID LEUKODYSTROPHY 1		MVB	A miasmic leukodystrophy disease. It affects the growth of the myelin sheath, the fatty covering (which acts as an insulator) on nerve fibers in the Central Nervous System. The disease creates a lack of transporter protein (now numbered SC16A2) for the thyroid hormone, triiodothyronine (T3) needed for the growth of nerve cells and nerve synapses. Constant tiredness, muscle weakness, weight and mental difficulties persist in adults. In children hypotonia, mental deficiencies, joint deformities (called contractures), muscle stiffness and involuntary movement of arms and legs. Often the children become wheelchair bound by adulthood. The disease was discovered in 2003/2004. T-2 deficiencies affect the brain. AKA PELIZAEUS-MERZBACHER DISEASE - CLASSIC
UCD ARG	UREA CYCLE DISORDER ARGINASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. These patients develop progressive spasticity. They can also develop tremor, ataxia, and choreoathetosis. Vibrational Arginase is available to help reverse the condition.
UCD ASL	UREA CYCLE DISORDER ARGININOSUCCINASE ACID LYASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. This disorder is marked by chronic hepatic enlargement and elevation of transaminases. Vibrational Argininosuccinic Acid Lyase is available to help reverse the condition. It is often treated with only arginine supplement.
UCD ASS	UREA CYCLE DISORDER ARGININOSUCCINIC ACID SYNTHASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. It is nick-named Citrullinemia. Some forms block bile and some are most known in Japanese populations. Vibrational Citrulline is used to help the reversal.
UCD CPS	UREA CYCLE DISORDER CARBAMOYL PHOSPHATE SYNTHASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. Vibrational Carbamoyl Glutamate is available to help the reversal. This form usually results in childhood death unless intercepted. No known adult symptoms are identified (see childhood symptoms).
UCD NAG	UREA CYCLE DISORDER N-ACETYLGLUTAMATE SYNTHETASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. This later-onset form is associated with intense headaches, partial loss of vision, problems with balance and muscle coordination (ataxia), and lethargy. Vibrational citrulline is available to help reverse the condition.
UCD OTC	UREA CYCLE DISORDER ORNITHINE TRANSCARBMYLASE		MVB	Urea Cycle Disorders are a group of genetic liver disorders of missing or deficient enzymes required to synthesize nitrogen from digested protein. In the absence of these enzymes nitrogen is not converted to urea, but instead leaves a residue of ammonia in the blood which invades the central nervous system, brain and blood. Digestive disorders, hepatomegaly, respiratory alkalosis, brain damage and central nervous system deficiencies result. The diseases are most noted in childhood and are now being recognized in adulthood. The name of the remedy identifies the missing enzyme. This is the first step of the urea cycle. Hyperactivity and various forms of aggression start in childhood and carry through to adulthood. The condition is most manifest in boys, although evidence is appearing of respiratory alkalosis and poor appetite in women over 60. Vibrational citrulline and vibrational arginine are available to help the reversal.